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来源：百度知道 编辑：UC知道 时间：2024/06/04 16:01:32
目的 研究三甲基氯化锡（TMT）对体外脉络丛NKATP酶活力的影响，并为探讨TMT中毒引起中枢神经系统损伤的机制提供体外实验证据。方法 参照ATP酶试剂盒测定方法，分别设定10、50、250、500、800μM/L的TMT和Ouabain实验管，设定空白对照管和TMT、Ouabain相应剂量的对照管，每组平行三份共66管，分别测定各管NKATP酶活力。结果 不同浓度的TMT 和Ouabain实验组NKATP酶活力均减小，差异有统计学意义（P<0.01或P<0.05）。结论 各浓度的TMT和Ouabain对体外脉络丛NKATP酶活力均有抑制作用，TMT的抑制作用有剂量反应关系，因此TMT中毒引起中枢神经系统损伤的体外机制成立
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Objective To study the trimethyltin chloride (TMT) on the in vitro choroid plexus NKATP enzyme activity, and to explore the TMT poisoning caused by the central nervous system injury mechanism in vitro evidence. Methods ATP enzyme kit method, respectively, set 10,50,250,500,800 μM / L of TMT and Ouabain experimental tubes, pipes and control settings TMT, Ouabain corresponding dose of control tubes, each of the three parallel A total of 66, were measured by enzyme activity NKATP the tube. The results of different concentrations of TMT and Ouabain experimental group NKATP enzyme activity decreased, the difference was statistically significant (P <0.01 or P <0.05). Conclusion of the concentration of TMT and Ouabain on NKATP choroid plexus in vitro inhibit the enzyme activity, TMT inhibition in a dose-response relationship, so TMT poisoning caused by the central nervous system injury in vitro mechanisms for the establishment of